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Antipsychotic Drugs: Mechanisms of Action and Clinical Uses

Antipsychotic Drugs work by blocking dopamine receptors, particularly D2 receptors, in the brain. Dopamine is a neurotransmitter that plays a key role in psychosis. By blocking its receptors, antipsychotics reduce dopamine's excitatory effects and balance out its activity. The dopamine receptor D2 is an important mediator of neurological and behavioral functions and it is implicated in psychosis when it overstimulated. Antipsychotics decrease dopamine receptor stimulation, which in turn reduces psychotic symptoms.

In addition to blocking dopamine receptors, some antipsychotics also block serotonin receptors, histamine receptors, and alpha-adrenergic receptors. This multi-receptor action contributes to their effectiveness against psychosis as well as side effects. Newer "atypical" antipsychotics are less likely than older drugs to block dopamine and serotonin receptors, providing similar effectiveness against psychosis with a lower risk of extrapyramidal symptoms.
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